PR Firm Promoting Alzheimer’s Disease Prevention
Neurological diseases known collectively as dementia are the fastest-growing cause of death in the world. The epidemic is spreading exponentially because of global misinformation and mismanagement. Patients, caregivers, family members and millions of other stakeholders deserve the truth.
Only a decade ago, the idea that Alzheimer’s disease might be transmissible between people would have been laughed away. But scientists have now shown that tissues can transmit symptoms of the disease between animals. A new study published in the journal Nature raises additional concern about the transmissibility of Alzheimer’s disease between people. The impact of such research is profound.
“This is the first evidence of real-world transmission of amyloid pathology,” says molecular neuroscientist John Hardy of University College London (UCL). “It is potentially concerning.”
Most of us know dementia as Alzheimer’s disease, Parkinson’s disease and Creutzfeldt-Jakob disease. They are all part of the same disease spectrum. It’s negligent not to treat them all as extremely transmissible diseases.
“There has been a resurgence of this sort of thinking, because there is now real evidence of the potential transmissibility of Alzheimer’s,” says Thomas Wiesniewski M.D. a prion and Alzheimer’s researcher at New York University School of Medicine. “In fact, this ability to transmit an abnormal conformation is probably a universal property of amyloid-forming proteins (prions).”
Studies confirm that people and animals dying of prion disease contaminate the environment around them with prions because prions are in the urine, feces, blood, mucus and saliva of each victim. Not only are homes and hospitals exposed to the prion pathogen, so are entire sewage treatment systems and their by-products. Wastewater treatment plants are prion incubators and distributors.
Dementia is vastly undiagnosed and misdiagnosed. Unfortunately, doctors are withholding millions of additional diagnoses, so we don’t know the extent of the epidemic. Mismanagement on many levels is an outrage.
For example, former U.S. President Ronald Reagan died in 2004 after a long battle with Alzheimer’s disease. His death certificate, however, listed pneumonia as the cause of death. Attributing Alzheimer’s deaths to other causes is common. Such practices are masking the body count with labels. The actual numbers are staggering and they will continue to escalate. The burden on unprepared families is surging.
Despite underreporting, we know that about 50 million people around the world already have Alzheimer’s disease and other forms of dementia. Millions of other victims have already died. The global burden of dementia care in 2015 is estimated at $818 billion (up from $214 billion in 2010).
Transmissible Spongiform Encephalopathy
So-called “Alzheimer’s disease” and closely related diseases are actually members of an aggressive family of neurodegenerative diseases known as transmissible spongiform encephalopathy (TSE). The operative word is “transmissible.” The TSE epidemic represents an environmental nightmare that threatens every mammal on Earth.
According to research from John Hopkins, Duke University, and Utah State University, caregivers of someone with dementia are six times more likely to develop the condition themselves.
TSEs include Alzheimer’s disease, Creutzfeldt-Jakob disease, Parkinson’s disease, mad cow disease and chronic wasting disease in deer. TSE has been found in mink, moose, mice, sheep, cats, elephants, dolphins and many other species. Sea mammals are extremely vulnerable, but they aren’t being tested. Sick mammals on land and at sea are a canary in a coal mine. Their sickness confirms an alarming epidemiological trend among humans. An environmental contagion is responsible for the spike among many mammals. There is no species barrier.
TSEs are caused by a deadly protein called a prion (PREE-on). Prion disease is unstoppable. The pathogen spreads through the bodily fluids and cell tissue of its victims. Blood, saliva, mucus, milk, urine and feces carry deadly prions from victims. All tissue is infectious just because of the contact with the contaminated blood.
Dr. Stanley Prusiner, an American neuroscientist from the University of California at San Francisco, earned a Nobel Prize in 1997 for discovering and characterizing deadly prions and prion disease. He claims that all TSEs are caused by prions. Prions are a deadly and unstoppable form of protein that migrates, mutates, multiplies and kills with unparalleled efficiency.
President Obama awarded Prusiner the National Medal of Science in 2010 to recognize the importance of his research. According to Prusiner, TSEs all are on the same disease spectrum, which is more accurately described as prion (PREE-on) disease Creutzfeldt-Jakob disease is at the extreme end of the spectrum. Prusiner’s science is being ignored and we are facing a public health disaster because of the negligence.
Prions are such a formidable threat that the U.S. government enacted the Bioterrorism Preparedness and Response Act of 2002, which included a provision to halt research on prions in all but two laboratories. It classified prions as select agents that pose an extreme risk to food, water and health systems. Unfortunately, the Center For Disease Control quietly took prions off the list about two years ago because the classification threatened to criminalize some multi-billion dollar industries and many industry practices.
The Problem With Prions
Prions linger in the environment, homes, hospitals, nursing homes, dental offices, restaurants and many other places infinitely. They migrate, mutate, multiply and kill with unparalleled efficiency. Prions defy all attempts at sterilization and inactivation. Victims often become infectious long before they appear sick.
“The (human) brain diseases caused by prions include Alzheimer’s, Parkinson’s, Huntington’s, amyotrophic lateral sclerosis (Lou Gehrig’s disease), and other disorders known as frontotemporal dementias,” said Nobel Laureate Stanley Prusiner.
Due to many factors, prion disease is a spectrum disease. Alzheimer’s disease and Parkinson’s disease are the most common human forms of prion disease. Alzheimer’s and Creutzfeldt Jakob disease (CJD) are the common diagnoses when the primary symptom is dementia. Parkinson’s is the common diagnoses when the primary symptom is a movement disorder. Some victims exhibit both symptoms.
“CJD behaves like Alzheimer’s disease on steroids,” said Dr. Jennifer Majersik, an associate professor of neurology at the University of Utah.
One reason for the variations of prion disease is that there are many mutations of prions. More mutations are created every day as prions migrate throughout the food chain and the environment. Prions shed from humans, for example, are the most deadly. As such, some prions can kill people within weeks of exhibiting clinical symptoms, while others can take years. Other people may not fall victim to the disease, but they can serve as carriers of the pathogen (internally and externally).
Since prion disease is a spectrum disease, doctors can’t tell the difference between Alzheimer’s disease and CJD without a spinal tap. Unfortunately, there are so many variations of prions, that even a spinal tap can only detect prion disease. It can’t distinguish between all of the various mutations because they aren’t even in the science books, yet. Dr. Prusiner just announced the discovery of a new form of prion. The first such characterization in 40 years. Just a few thousand more to go. Unfortunately, it’s time to focus on containment not characterization.
For most doctors, diagnosing neurological disease is a process of elimination and a shot in the dark. As such, diagnoses are wrong at least 20 percent of the time. The only definitive diagnosis of prion disease requires an autopsy, which rarely happens with neurological disease due to concerns about deadly contamination and exposure. All doctors are guessing with each Alzheimer’s diagnosis. All they can do is attempt to gauge the severity of symptoms. This problem complicates the search for accurate statistics about the size and scope of the epidemic. The attempt to differentiate between CJD and Alzheimer’s represents a containment nightmare.
Alzheimer’s Disease Caregivers Misinformed
As stated before, blood, saliva, mucus, aerosols, milk, urine, and feces of victims carry deadly prions. These bodily fluids and tissues create biohazards in the homes and communities of victims.
Unfortunately for caregivers and family members, this is a deadly issue. The protocol for patient care and caregiver safety is vastly different for Alzheimer’s and Parkinson’s disease patients versus CJD patients. Only CJD patients have been treated as infectious until now. It’s time for immediate changes to account for risks associated with those battling Alzheimer’s disease and Parkinson’s disease. The double standard demonstrates incompetence and negligence. Such reckless policies have exposed millions of people to a highly contagious neurological disease.
Although there are many causes and pathways contributing to the prion disease epidemic, many pathways are being mismanaged around the globe. Not only are homes exposed to the prion pathogens from victims, so are entire sewage systems.
Via sewage, biosolids, and reclaimed sewage water, we’re recycling the prion pathogen from victims right back into our food and water supplies. We’re dumping these killer proteins on crops, parks, golf courses, gardens, ski areas, school grounds and beyond. Wind, rain and irrigation spread them throughout our communities and watersheds.
Since prions are in the bodily fluids of its victims, sewage mismanagement is a top concern. Sewage treatment plants can’t detect or stop prions. Just ask the U.S. EPA. Therefore, putting biosolids on parks, crops and golf courses is a very bad idea. Dumping sewage from billions of people on land and at sea spreads the prion pathogen far and wide. The original risk assessments prepared by the U.S. UPA were severely flawed. Many risks were not addressed at all, including prions. Failure to update those risk assessments and the land application policies that rely on them is negligent. Several nations around the world, including Canada, follow the U.S. EPA’s guidance and risk assessments on biosolids.
Based on these facts, water reuse (reclaimed wastewater) for drinking water is reckless. Using sewage sludge and biosolids on crops, grazing land, lawns and gardens is foolish and fraudulent. It’s been proven that plants grown in sewage sludge absorb prions and they become infectious.
Every sewage system in the world has been used by a person, if not millions of people, with Alzheimer’s disease, Parkinson’s disease and Creutzfeldt-Jakob disease. Sewage systems have become prion incubators. Biosolids, water reuse and sludge disposal have made them prion distributors. Sewage mismanagement is exposing millions of people, livestock, wildlife, crops and water supplies to deadly neurological disease.
We’re all vulnerable to Alzheimer’s and other forms of prion disease right now due to widespread denial and mismanagement. As a result, the average age of people diagnosed with Alzheimer’s and CJD is dropping. The autism epidemic continues to surge. It’s all related.
Caregivers, family members and others living with, or near, Alzheimer’s and CJD patients are at risk of transmission of the disease. Although there are multiple causes of prion disease, reckless policies around the world are contributing to an unstoppable environmental pathogen. It’s spreading further every day. Please join us and advocate for the truth and reform. Read More About The Connection Between Sewage Sludge, Alzheimer’s Disease and Autism. http://alzheimerdisease.tv/autism/